About: Molecular and cellular mechanisms in the viral exacerbation of asthma

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About: Molecular and cellular mechanisms in the viral exacerbation of asthma Goto Sponge NotDistinct Permalink

An Entity of Type : schema:ScholarlyArticle, within Data Space : covidontheweb.inria.fr associated with source document(s)

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type	Academic Article research paper schema:ScholarlyArticle
isDefinedBy	Covid-on-the-Web dataset
title	Molecular and cellular mechanisms in the viral exacerbation of asthma
Creator	Mahalingam, Suresh Schwarze, Jürgen Tauro, Sharyn Tripp, Ralph Matthaei, Klaus »more»
source	Elsevier; Medline; PMC
abstract	The aetiology of asthma associated with viral infection is complex. The dynamics that contribute to disease pathogenesis are multifactorial and involve overlapping molecular and cellular mechanisms, particularly the immune response to respiratory virus infection or allergen sensitization. This review summarizes the evidence associated with factors that may contribute to the development or exacerbation of asthma including age, host factors, genetic polymorphisms, altered immune responses, and aspects of viral antigen expression. This review also provides an important perspective of key events linked to the development of asthmatic disease and related pulmonary inflammation from human and animal studies, and discusses their relationship as targets for disease intervention strategies.
has issue date	2008-08-13(xsd:dateTime)
bibo:doi	10.1016/j.micinf.2008.07.037
bibo:pmid	18762266
has license	no-cc
sha1sum (hex)	8ba87cfd768755edda2df37edc26d3db72d4a579
schema:url	https://doi.org/10.1016/j.micinf.2008.07.037
resource representing a document's title	Molecular and cellular mechanisms in the viral exacerbation of asthma
has PubMed Central identifier	PMC7172315
has PubMed identifier	18762266
schema:publication	Microbes Infect
resource representing a document's body	covid:8ba87cfd768755edda2df37edc26d3db72d4a579#body_text
is schema:about of	named entity 'disease' named entity 'events' named entity 'pulmonary' named entity 'cellular' named entity 'TARGETS' »more»

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